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Diabetic Wounds
Home » Diabetic Wounds
Diabetic Wounds
Home » Diabetic Wounds
Diabetic Wounds
What Are Diabetic Wounds?
Diabetic Wounds are primarily associated with diabetic ulcers, which are open sores that can develop due to complications from diabetes.
These wounds often arise from minor injuries that go unnoticed due to nerve damage (neuropathy) and can lead to severe complications if not treated promptly.
Symptoms
Diabetic wounds may present with various symptoms, including:
-
Redness or Swelling:
The area around the wound may appear inflamed.
-
Drainage:
lear or colored pus may be present, indicating infection.
-
Pain or Discomfort:
Although neuropathy may reduce pain sensation, some individuals may still experience discomfort.
Treatments
Shockwave Spark Wave® Therapy
- Can trigger transient activation of proinflammatory pathways
- Enhances cell proliferation by acting on ATP release.
- Leads to a cytoprotective response.
- Supports tissue regeneration.
Procellera
- Generate electricity designed to mimic the skin’s electrical energy.
- Demonstrated to kill existing biofilm and prevent biofilm from forming (in vivo pre-clinical model).
- Demonstrated to increase the rate of cell migration and re-epithelialization.
Treatments
Shockwave Spark Wave® Therapy
- Can trigger transient activation of proinflammatory pathways
- Enhances cell proliferation by acting on ATP release.
- Leads to a cytoprotective response.
- Supports tissue regeneration.
Procellera
- Generate electricity designed to mimic the skin’s electrical energy.
- Demonstrated to kill existing biofilm and prevent biofilm from forming (in vivo pre-clinical model).
- Demonstrated to increase the rate of cell migration and re-epithelialization.
Shockwave Spark Wave® Therapy
- Can trigger transient activation of proinflammatory pathways
- Enhances cell proliferation by acting on ATP release.
- Leads to a cytoprotective response.
- Supports tissue regeneration.
Procellera
- Generate electricity designed to mimic the skin’s electrical energy.
- Demonstrated to kill existing biofilm and prevent biofilm from forming (in vivo pre-clinical model).
- Demonstrated to increase the rate of cell migration and re-epithelialization.